virology · step 1

Medical Virology · Step 1 Notes

Viral architecture, hepatitis, DNA/RNA viruses, herpes, antivirals & high-yield pearls

🧬 Viral architecture & life cycle

Key concept: viruses are obligate intracellular parasites. Their structure determines stability, tropism, and replication strategy.

Naked vs. enveloped

  • Naked (non-enveloped): resist detergents, acid, drying; often released by lysis. Examples: adenovirus, parvovirus, papillomavirus.
  • Enveloped: fragile; lipid envelope contains viral glycoproteins essential for attachment. Bud from host membranes. Examples: herpesvirus, influenza, HIV.

Genome types

  • dsDNA – most DNA viruses (except parvovirus).
  • ssDNA – parvovirus B19.
  • +ssRNA – can directly serve as mRNA (picornaviruses, flaviviruses).
  • −ssRNA – require virion-associated RNA-dependent RNA polymerase.
  • dsRNA – reoviruses.
  • Retroviruses – +ssRNA with reverse transcriptase.

General replication steps

Attachment Entry Uncoating Macromolecular synthesis Assembly Release
🔬 High-yield: Enveloped viruses acquire their envelope by budding; naked viruses cause cell lysis. Persistent infections are more common with enveloped viruses (e.g., HBV, HIV).

🩺 Viral hepatitis (A–E)

Hepatitis viruses are hepatotropic but differ in transmission, chronicity, and outcomes.

VirusFamily / genomeTransmissionChronic?Key features
HAVPicornavirus, +ssRNA, nakedFecal–oralNoMild acute; no sequelae. IgM anti-HAV = acute.
HBVHepadnavirus, dsDNA (partially), envelopedParenteral, sexual5–10% adults, 90% infantsCirrhosis, HCC. HBsAg, anti-HBc IgM.
HCVFlavivirus, +ssRNA, envelopedParenteral~80%Often subclinical → chronic, HCC. Anti-HCV.
HDVViroid-like, circular RNA, enveloped (HBsAg)ParenteralCo-infection/superinfectionRequires HBV. Severe fulminant.
HEVHepevirus, +ssRNA, nakedFecal–oralRare (immunocompromised)Severe in pregnancy (25% mortality).
🧪 HBV serology: HBsAg positive = acute or chronic infection. Anti-HBc IgM = acute. Anti-HBs = recovery/vaccination. HBeAg = high infectivity.

🧫 DNA viruses – overview

Most are dsDNA, icosahedral, replicate in the nucleus (poxvirus exception: cytoplasm).

FamilyEnvelopeGenomeReplication siteClinically relevant
ParvoviridaeNakedssDNANucleusB19 (fifth disease, aplastic crisis)
PapillomaviridaeNakeddsDNA circularNucleusHPV (warts, cervical cancer)
PolyomaviridaeNakeddsDNA circularNucleusBK, JC (PML)
AdenoviridaeNakeddsDNA linearNucleusARD, conjunctivitis, gastroenteritis
HepadnaviridaeEnvelopeddsDNA (partial)Nucleus (RNA intermediate)HBV
HerpesviridaeEnvelopeddsDNANucleusHSV, VZV, EBV, CMV, HHV-6, HHV-8
PoxviridaeEnvelopeddsDNACytoplasmVariola, molluscum, monkeypox

▶ Parvovirus B19

  • Disease: Fifth disease (erythema infectiosum) – “slapped cheek” rash in children; aplastic crisis in sickle cell; hydrops fetalis.
  • Pathogenesis: targets erythroid progenitors.

▶ HPV (Papillomaviridae)

  • Types 6,11 – genital warts (benign).
  • Types 16,18 – cervical, anal, oropharyngeal cancer. E6/E7 inactivate p53/Rb.
  • Diagnosis: koiocytes on Pap smear.

🦠 Herpesviridae (8 human types)

Large dsDNA, enveloped, establish latency, reactivation.

HSV-1 & HSV-2

  • HSV-1: oral cold sores, keratitis, encephalitis (temporal lobe). Latent in trigeminal ganglion.
  • HSV-2: genital herpes, neonatal herpes. Latent in sacral ganglia.
  • Treatment: acyclovir (requires viral thymidine kinase).

VZV

  • Primary = chickenpox (asynchronous rash). Reactivation = shingles (dermatomal).
  • Latent in dorsal root ganglia.

EBV

  • Infectious mononucleosis: fever, pharyngitis, lymphadenopathy, splenomegaly.
  • Heterophile-positive (Monospot). Atypical lymphocytes (Downey cells).
  • Associated with Burkitt lymphoma, nasopharyngeal carcinoma.

CMV

  • Heterophile-negative mononucleosis. Congenital infection: jaundice, petechiae, “blueberry muffin” baby.
  • Owl-eye inclusions. Retinitis in AIDS.

HHV-6 / HHV-8

  • HHV-6: roseola (exanthem subitum) – fever then rash.
  • HHV-8: Kaposi sarcoma (AIDS-related).

📌 Poxviridae & other DNA viruses

Smallpox (variola): eradicated; synchronous rash, same stage. Guarnieri bodies (cytoplasmic).
Molluscum contagiosum: umbilicated warts; eosinophilic inclusion bodies.

Monkeypox (2022 outbreak)

  • Transmission: respiratory, direct contact. Rash, lymphadenopathy.
  • Highest risk: men who have sex with men (during outbreak).

💊 Antiviral drugs – mechanisms

StageDrug exampleTarget / mechanismVirus
AttachmentMaravirocCCR5 antagonistHIV
PenetrationEnfuvirtideFusion inhibitorHIV
UncoatingAmantadineM2 blocker (influenza A)Influenza
Nucleic acid synthesisAcyclovir, zidovudineDNA polymerase / RT inhibitionHSV, HIV
ProteaseRitonavirProtease inhibitorHIV
IntegraseRaltegravirIntegrase inhibitorHIV
ReleaseOseltamivirNeuraminidase inhibitorInfluenza
📌 Exam pearl: Acyclovir requires viral thymidine kinase for activation; resistance often due to TK mutations.

🧪 RNA viruses – general features

  • Most are enveloped (exceptions: picornavirus, calicivirus, hepevirus, reovirus).
  • Segmented genomes: orthomyxovirus, bunyavirus, arenavirus, reovirus.
  • −ssRNA viruses carry RNA-dependent RNA polymerase in the virion.
+ssRNA → directly translated (e.g., poliovirus, HCV).
−ssRNA → requires polymerase to make mRNA (e.g., influenza, rabies).
Retroviruses → reverse transcriptase (RNA → DNA).

⚡ Clinical pearls & high-yield

  • Encephalitis: HSV-1 is the most common cause of sporadic viral encephalitis in the U.S. (temporal lobe involvement).
  • Mononucleosis: heterophile-positive = EBV; heterophile-negative = CMV.
  • Immunocompromised: CMV retinitis, HHV-8 Kaposi, JC virus PML.
  • Hepatitis B: window period = HBsAg disappears, anti-HBs not yet detectable; anti-HBc IgM positive.
  • HPV vaccine: Gardasil 9 covers types 6,11,16,18,31,33,45,52,58.
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